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We can’t base public health policy on one MP’s experience


Whilst I agree Ellie that to lose fat mass then the body needs to reduce its stored calories (specifically fat stores), we must remember a human is a complex system which includes multiple hormones that can affect fat storage or fat mobilisation. The food we eat, and the various macronutrients and their amounts in a diet, can help or hinder the control systems that relate to fat storage and also to glucose homeostasis. Here's a small collection of thoughts on this. 1. Insulin resistance + incoming glucose (sugar or starch) == hyperinsulinaemia. A clinical recommendation... measure the fasting insulin level of a patient with markers of insulin resistance (e.g. The metabolic syndrome criteria) and then give them a glucose load of 75g, and remeasure their insulin at 1 hour and 2 hours. Then consider how this information (hyperinsulinemic response) can help in shared decision making and the application of evidence based medicine in what dietary approach may be most effective and enjoyable for the patient. (Joseph Kraft's work on this is insightful... His book "The Diabetes Epidemic and You" is a good read). 2. Insulin's actions include promoting fat storage, and reducing fat mobilisation. Obesity is reduced when the amount of fat mobilised from stores is greater than the amount of fat moved into stores. Thus a lower insulin level would seem helpful for reducing obesity. 3. There is a general consensus that "low carbohydrate" means

Posted date

29 Sep 2018

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